Joint and muscle problems
associated with fungal exposure; the full explanation |
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Fibromyalgia is the diagnosis doctors give when autoimmune symptoms
develop at the same time the patient suffers from sore joints,
painful muscles, disturbed microcirculation, chronic fatigue,
impaired immunity, sleep disorders, and a reduced level of energy.
(Lancet, 1992). It is difficult to identify the causes, triggers and
origins of Fibromyalgia, and is often misdiagnosed, and some
physicians are still uneducated and ignorant about this debilitating
disease. But make no
mistake the disability is progressive and destructive especially to
the high proportion of women that develop Fibromyalgia. In
simple terms, mold patients of suffer from loss of synovial fluid
around their joints, resulting to loss of lubrication. Another
hypotheses is that in addition to this joint problem, is the
symptomology of lack of fluid in the fascia; the connective tissue
throughout the body. Unfortunately, many
pharmadocs are lost at recognizing, diagnosing, or treating this
illness since they do not know what tests to conduct and in most
cases it is not treated with drugs. This makes fibromyalgia
exceedingly frustrating for the medical community.
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Early symptoms of Fibromyalgia and
Myofascial Pain Syndrome:
"(FMS)" indicates that the
preceding symptom often accompanies fibromyalgia. "(H)" indicates
that reactive hypoglycemia or insulin resistance may contribute to
the symptom. The Latin-derived names of the muscles in brackets [ ]
indicate the most likely muscles that may have trigger points that
could cause the symptom. The symptoms are listed in boldface type,
but by design, they are not in any particular order. There may be a
variety of causes for every symptom, and there are many ways of
categorizing the symptoms. I do not wish to imply connections where
there may not be any, nor, by separating symptoms, to exclude them
where they may exist. Each of us is different and our patterns of
symptoms may be different. You may notice that the diagnostic
criteria for FMS and MPS are very similar to the symptoms of fungal
exposure.
"Traveling" nocturnal sinus
stuffiness: [pterygoid, sternocleidomastoid, posterior
digastric]
Unexplained allergies: (FMS)
Post nasal drip: (FMS), [pterygoid,
sternocleidomastoid]
Drooling in sleep:
[internal medial pterygoid]
Swollen glands: [digastric]
Difficulty swallowing: [digastric,
pterygoid]
Dry cough: [lower end
sternal sternocleidomastoid]
TMJ symptoms: [masseter,
trapezius, temporalis, pterygoid]
Dizziness when turning head or
changing field of view: [sternocleidomastoid], (H)
Runny nose: (FMS), [sternocleidomastoid,
pterygoid]
Sore throat: [sternocleidomastoid,
digastric, pterygoid]
Stiff neck: [levator
scapulae]
Mold/yeast sensitivity:
(FMS), (H)
Reflux esophagitis:
[external oblique], (H)
Headaches/migraines:
(FMS), [trapezius, sternocleidomastoid, temporalis, splenii,
suboccipital, semispinalis capitis, frontalis, zygomaticus major,
cutaneous facial, posterior cervical], (H)
Light and/or broken sleep pattern
with unrefreshing sleep: (FMS)
Sweats: (FMS), (H)
Morning stiffness: (FMS),
[multiple TrPs]
Fatigue: (FMS), [multiple
TrPs], (H)
Shortness of breath: (FMS)
[serratus anterior, diaphragm, other respiratory muscles], (H)
Painful weak grip that may let go:
[infraspinatus, scaleni, hand extensors, brachioradialis]
Menstrual problems and/or pelvic
pain: (FMS), [coccygeus, levator ani, obturator internus,
high adductor magnus, abdominal obliques]
PMS: (FMS)
Loss of libido: (FMS)
Low back pain: [quadratus
lumborum, thoracolumbar paraspinals, longissimus, ilicostalis,
multifidi, rectis abdominis]
Nail ridges and/or nails that curve
under: (FMS)
Difficulty speaking known words:
(FMS), (H)
Directional disorientation:
(FMS), (H)
Visual perception problems:
[sternocleidomastoid], (H)
Tearing/reddening of eye, drooping
of eyelid: [upper sternal sternocleidomastoid]
Loss of ability to distinguish some
shades of colors: (FMS)
Short-term memory impairment:
(FMS), (H)
Weight gain/loss: (FMS),
(H)
Sensitivity to odors:
(FMS)
Mitral valve prolapse:
(FMS)
Double/blurry/changing vision:
[internal eye muscles, temporalis, sternocleidomastoid, trapezius,
cutaneous facial, splenius cervicis]
Visual and audio effects/falling
sensations before sleep (called "sleep starts"): (FMS)
Earaches/ringing/itch:
(FMS), [SCM, masseter, pterygoid],
Unexplained toothaches: [temporalis,
masseter, digastric]
Rapid/fluttery/irregular
heartbeat/heart attack-like pain: (FMS), [sternalis,
pectoralis], (H)
Bloating/nausea/abdominal cramps:
(FMS), [abdominals, multifidi, iliocostalis, paraxiphoid
rectus abdominus, quadratus lumborum, upper thoracic paraspinals],
(H) [Note: for excessive gas and belching, check for TrP at angle of
12th rib, either side.]
Appendicitis-like pains: [iliopsoas,
rectus abdominis, piriformis, iliocostalis]
Carbohydrate/chocolate cravings:
(FMS), (H)
Sensitivity to
cold/heat/humidity/pressure changes/light/wind: (FMS)
Abdominal cramps, colic: [periumbilical
rectus abdominus]
Panic attacks: (FMS), (H)
Mottled skin: (FMS)
Depression: (FMS), (H)
Confusional states: (FMS),
(H)
Thumb pain and tingling numbness:
[brachialis entrapment of radial nerve, adductor pollicus]
Urine retention: [upper
pubic, inguinal ligament, lower internal oblique and lower rectus
abdominus TrPs]
Tendency to cry easily:
(FMS), (H)
Night driving difficulty:
(FMS)
Weak ankles: [peroneus,
tibialis]
Lax, pendulus abdomen:
[abdominal TrPs, especially in rectus abdominus]
Upper/lower leg cramps: [sartorius,
gastrocnemius]
Tight Achilles tendons: [tibialis
posterior]
Groin pain: [adductores
longus and brevis, iliopsoas]
Irritable
bowel: (FMS), [pelvic TrPs,
multifidi, high adductor magnus, abdominal obliques], (H)
Sciatica: [thoracolumbar
paraspinals, gluteus minimus, hamstrings, piriformis, iliopsoas]
Urinary frequency: (FMS),
[cutaneous and myofascial lower abdominal TrPs]
Impotence: (FMS), [piriformis
pudendal nerve entrapment]
Stress incontinence, anal/genital/perineal
pain: [pelvic floor TrPs, high adductor magnus, piriformis,
paraspinals]
Painful intercourse:
[vaginal TrPs, pelvic floor TrPs, piriformis]
Muscle twitching: (FMS),
[local TrPs]
Numbness and tingling:
[nerve entrapment by TrPs]
Diffuse swelling: (FMS),
[vascular entrapment by TrPs]
Hypersensitive nipples/breast pain:
[pectoralis]
Fibrocystic breasts:
(FMS), [possible ductile entrapments by TrPs]
Buckling knee: [vastus
medialis, quadriceps, adductor longus]
Problems climbing stairs:
[sartorius, quadriceps femoris, vastus medialis]
Problems going down stairs:
[popliteus]
Free-floating anxiety:
(FMS), (H)
Mood swings: (FMS), (H)
Unaccountable irritability:
(FMS), (H)
Trouble concentrating:
(FMS), (H)
Shin splint-type pain: [peroneus,
tibialis]
Heel pain: [soleus,
quadratus plantae, abductor hallucis, tibialis posterior]
Sensory overload: (FMS),
(H)
Handwriting difficulties:
[adductor/opponens pollicis]
Sore spot on top of head:
[splenius capitis, sternocleidomastoid]
Problems holding arms up (as when
folding sheets): [subscapularis, infraspinatus,
supraspinatus, upper trapezius, levator scapulae]
"Fugue"-type states (staring into
space before brain can function): (FMS), (H)
Tight hamstrings:
[hamstring complex, adductor magnus, quadriceps femoris, iliopsoas,
gastrocnemius] Numbness/tingling on the outer thigh (meralgia
paresthetica): [quadriceps femoris, vastus lateralis, sartorius,
tensor fascia latae entrapment]
Carpal tunnel-like pain in wrist
(watchband area): [subscapularis]
Balance problems/staggering gait:
[sternocleidomastoid, gluteus minimus], (H)
Restless leg syndrome: [gastrocnemius,
soleus]
Myoclonus (muscle movements and
jerks at night): (FMS), [local TrPs]
Feeling continued movement in car
after stopping: [sternocleidomastoid]
Feeling tilted when cornering in
car: [sternocleidomastoid]
First steps in the morning feel as
if walking on nails: [long flexors of toes, tibialis
posterior]
Pressure of eyeglasses or headbands
is painful: [head, neck and shoulder TrPs]
Thick secretions: (FMS)
Bruise/scar easily: (FMS)
Some stripes and checks cause
dizziness: [sternocleidomastoid]
Bruxism (teeth grinding):
(FMS), [digastric, masseter, soleus]
Inability
to recognize familiar surroundings:
(FMS), (H)
Delayed reactions to "overdoing
it": (FMS)
Tissue overgrowth (fibroids,
ingrown hairs, heavy and splitting cuticles, adhesions):
(FMS)
A myofascial trigger point is a
localized area starving for oxygen. It creates an increased local
energy demand. This local energy crisis releases neuroreactive
biochemicals which sensitize nearby nerves. The sensitized nerves
initiate the motor, sensory, and autonomic effects of myofascial
trigger points by acting on the central nervous system. Muscles with
trigger points are muscles in a constant state of energy crisis.
Myofascial trigger points can be identified and documented
electrophysiologically by characteristic spontaneous electrical
activity (SEA). They may also be identified histologically (which
means that the structure of the cells have changed) by contraction
knots — the lumps and bumps we know only too well. Both of these
phenomenon seem to result from excessive release of the
neurotransmitter acetylcholine (ACh) from the nerve terminal of the
motor endplate (the complex end formation of the nerve).
We now have objective confirmation of electromyographic imaging of a
myofascial trigger point. There is also ultrasound imaging of local
twitch responses of trigger points, and biopsies of myofascial
trigger points that show contraction knots and giant rounded muscle
fibers. To quote from this article, "The endplate dysfunction
characteristic of MTrPs involves both the nerve terminal and the
postjunctional muscle fiber. This relationship identifies MTrPs as a
neuromuscular disease." Simons DG. 1999. Diagnostic criteria of
myofascial pain caused by trigger points. J Musculoskeletal Pain
7(1-2):111-120.
A MTrP is always found in a taut
band which is histologically related to contraction knots caused by
excessive release of ACh in an abnormal endplate. The pathogenesis
of myofascial trigger points appears to involve serious disturbance
of the nerve ending and contractile mechanism at multiple
dysfunctional endplates. Doctor Hong has even formed a theory
concerning fibromyalgia tender points. Hong, C-Z. 1999. Current
research on myofascial trigger points-pathophysiological studies. J
Musculoskeletal Pain 7(1-2):121-129.
Please ask your librarian to obtain these articles through
Interlibrary loan, and give them to your doctor. Don’t forget to
keep copies for yourselves.
Common permanent symptoms of FMS
and MPS include:
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Temporomandibular
Joint Dysfunction Syndrome
Multiple Chemical Sensitivity Syndromes
Dizziness
Depression and anxiety (onset)
Irritable bowel syndrome
(beginning signs)
Skin complaints
Morning stiffness
Chronic headaches
Myofascial pain syndrome
“Fibrofog”: Cognitive or Memory Impairment
Chronic fatigue
- Abnormally high pain
sensitivity especially in the muscles and joints
- Insomnia or
unrefreshing sleep
- Chronic stiffness
especially in the shoulders, back and neck
- Reduced levels of
Human Growth Hormone
- Reduced levels of
ATP (Adenosine triphosphate) resulting in low or no energy
- Hormone and
endocrine imbalances
- Chronic toxin
accumulation from the skin, lungs, intestines, blood stream and
brain
- Depressed immune
function and activity
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*The
above symptoms can decrease immunity and allow or cause fibromyalgia
to worsen, producing additional symptoms that include: |
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- Depression and
personality changes
- PreMenstrual
Syndrome
- Tension and
migraine headaches
- Muscle trauma, and
deconditioning
- Anxiety, sleep
disorders vertigo, apathy, mood swings and memory loss
- Autoimmune
disorders like chemical hypersensitivity,
asthma, lupus and
arthritis
- Irritability
- Mitral valve
prolapse
- Opportunistic
bacterial, viral and parasite infections
- Chronic fatigue
immune deficiency
- Irritable bowel
syndrome (IBS) and leaky bowel syndrome
- Myofascial pain
syndrome
- Hypoglycemia and
inability to burn fat
- Intestinal and
food allergies
Causes of
Fibromyalgia
As the
number of contributing factors increase, the symptoms of
fibromyalgia increase until the body becomes weaker and less able to
identify self from non-self. Ultimately, the burden of multiple
problems decimate normal immune function, until the body can no
longer suppress the symptoms of fibromyalgia. It is interesting to
note that wherever the body is weakest the symptoms become
strongest. For this reason each fibromyalgia patient suffers in
different places. The scientific community will continue to discover
more specific causes for auto immune disorders but at present there
are four significant factors that have been proven to cause
fibromyalgia like symptoms, they are:
1. Fungal infections (primarily systemic)
2. Parasite/viral infestations
3. Bacterial infections
4. Chemical poisoning or prolonged exposure to chemical exposure
5. Severe trauma or shock
The viral, parasitic, and bacterial infections can trigger
autoimmune and hypersensitive reactions that may lead to auto immune
responses, but in most cases the human body is only exposed to that
infector a single time. The body can usually defend itself and
eliminate the disease or suppress all the symptoms. If there is only
one exposure the pathogen will be suppressed or it will mutate and
become a different manifestation. This is important because the
intestinal yeast infection can cause autoimmune disease that has
similar symptoms continuously, indicating that it is probably the
primary cause of fibromyalgia. A closer scientific review
demonstrates support for this theory.
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The results of this continual immune assault begins with pain
wherever the body is being attacked. The most common places for
autoimmune attacks occur in the muscles, soft tissues, joints or any
cell that is identified as a foreigner to the body. This includes
brain cells. When the immune system damages these tissues
sufficiently, it produces diseases like arthritis, irritable bowel
disease and
chronic fatigue syndrome.
Because
the body is at war with itself, the true cause of fibromyalgia must
be something that causes the body’s immune system to attack normal
tissues. In order for this to occur one or more of the following
must occur:
1. Normal immune cells that
identify normal tissue have become damaged.
2. Normal immune cells are sending improper messages.
3. The receptor sites on normal tissues have been damaged and cannot
receive proper messages.
4. Normal tissues are not sending or receiving normal messages.
5. Immune factors that communicate normal or attack status have been
adversely affected.
6. Something damaged the immune system, communicating immune
factors, or normal tissue receptor sites and is continuing to
interfere with normal immunity.
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Intestinal Yeast
(candida albicans),
and other fungal pathogens such as toxigenic molds, produce the
following toxins, which could be responsible for the symptoms and
causes of Fibromyalgia. This is not an all inclusive list.
1. Ethanol- an alcohol of intoxication mutates immune cells
2. Acetaldehyde 6 times more potent than ethanol, cell mutation
3. Tyramine interferes with immune function
4. Canditoxin interferes with and reduces immunity
5. Proteinase increases candida potency
6. Glycoprotein toxin interferes with immune function
7. Polysaccharride proteins reduces immunity
8. Histamine reduces immunity
9.
Mycotoxins that can cause
immune dysfunction and many other health problems
*Cyclosporin is a drug given to patients that have received
transplants. It prevents the rejection of the transplant by reducing
the function and activity of the immune system. With reduced
immunity the transplant is able to live and function within the
body. Cyclosporin has numerous adverse side effects including,
chronic fatigue, muscle and joint pain, opportunistic infections due
to lowered immunity, low energy, autoimmunity and a host of others
that mimic the definition of fibromyalgia. The important thing is
that the source of cyclosporin is a fungus found in Norway that is
very similar to candida albicans (yeast). Make no mistake one of the
primary causes of autoimmune syndromes like Fibromyalgia comes from
fungus cyclosporin, toxic mold, or candida albicans (intestinal
yeast).
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Reversing the Cause of Fibromyalgia |
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Most
fibromyalgia treatments merely treat the symptoms and never address
the cause.
Fibromyalgia is an autoimmune problem and the cause of the
autoimmunity must be stopped and reversed if a cure is to be
achieved. Since ethanol and acetaldehyde have been shown to cause
autoimmune responses and promote the growth of abnormal immune
cells, these toxins must be eliminated.
Note: The following do not heal or cure any type of disease. Only
the body can heal itself. However it is a well known fact that when
the body is given the proper nutrients to feed, cleanse, and protect
itself, it then has the ability to bring itself into balance and
combat the cause of autoimmunity and help restore proper immune
function and wellness.
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Lactobacillus acidophilus, bifodobacterium bifidum, lactobacillus
bulgarius, bifidobacterium longum, and streptococcus thermophilus.
Ensure it is without FOS.
These healthy intestinal flora are the primary tools for destroying
candida and restoring normal intestinal absorption and immune
function. Good flora works by secreting toxins that kill
candida
albicans. As candida retreats, the good flora re-establishes its
intestinal domain. This results in a reduction of the yeast and the
toxins it produces, which include ethanol and acetaldehyde. By
killing off the yeast and the toxins it produces, the following
benefits result:
A reduction in immune cell wall mutation, which reduces autoimmune
responses, which results in no more abnormal shaped immune cells
being reproduced. As the immune cells take two weeks to reproduce
normal cells, the body will cleanse for two weeks and then rebuild
steadily. Digestive gas bloating and irritability should subside
first, followed by a reduction in muscle and joint pain, followed by
a reduction in chemical sensitivities, followed by a continual
strengthening of the immune system, followed by improved health and
vigor and vitality.
This is the most important step in arresting the production and
replication of abnormal immune cells. This stops the cause of the
autoimmune response.
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a. Stop
eating refined sugars, fruit sugars, and start eating stevia
extract.
b. Stop eating milk sugars and dairy products
c. Avoid eating red meats, yeast, and caffeine
d. Avoid taking birth control pills, alcohol, and tobacco
e. Stop taking antibiotics for the problem
Following the
Mold Diet
has been proven to be very helpful.
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6.
Suggestions for the first phase of de-toxing:
a. Eat
correctly
b. Sleep 8 hours nightly
c. Supplement where there is a deficiency
d. Exercise and stretch daily, preferably in a warm pool.
e. Naturally hydrate with distilled or R/O water (8 glasses daily).
f. Cleanse the intestines and restore good flora daily. |
Update on fibromyalgia
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Expect a
dramatic cleansing of the fungi that usually results in a flu-like
illness that lasts about two weeks, after which a steady return to
good health begins. This return to good health takes at least one
month for mild cases, three to six months for moderate cases and six
to nine months for severe cases. Remember you are killing a fungal
infection that took years or decades to develop. Because of this it
may take months, and in some cases years to return to a normal
function. Virtually everyone that uses this protocol experiences
significant benefit and about half of the patients progress to a
point where they function normally without symptoms. At this point
it takes small continuous preventive amounts of these behaviors and
supplements to keep functioning normally. Following the
diet is an integral part of
effective detoxification. |
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1.
Lancet, 340(8827), 1992
2. Adv Exp Med Biol, 398, 373-379, 1996
3. Medical Hypothesis 44,369-378, 1995
4. Rheumatic Disease Clinics of North America 22(2), 219-243 1996
5. Current Opinion in Rheumatology 7(2),127-135, 1995
6. Clinical Rheumatology 15(3)283-286, 1996 200-205, 2003
7. Archives of Phys Med Rehab, 44, 369-378, 1995
This site is not intended to give medical advice. Seek
the advice of a professional for medication, treatment options, and complete
knowledge of any illness. The opinions expressed here are exclusively
my personal opinions do not necessarily reflect my peers or professional
affiliates. The information here does not reflect professional advice and is
not intended to supersede the professional advice of others.
Mold-Help. All
rights reserved-2001 - 2005® |
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